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Here we showed that imiquimod (IMQ) differentially regulates the expression of miR-17-92 cluster in the mouse skin, upregulating miR-17 and miR- 19 families and downregulating miR-92. The miR-17-92 cluster is often amplified or overexpressed in human cancers and has recently emerged as the prototypical oncogenic polycistron miRNA. The functional analysis of miR-17-92 is intricate by the existence of two paralogues: miR-106a-363 and miR-106b-25. Regulation of miRNA 219 and miRNA clusters 338 and 17-92 in oligodendrocytes Omar de Faria Jr. 1,2,3 , Qiao-Ling Cui 1,3 , Jenea M. Bin 1,2 , Sarah-Jane Bull 1,2 ,Timothy E. Kennedy 1,2 , The generation of myelinating cells in the central nervous system requires the initiation of specific gene expression programs in oligodendrocytes. We reasoned that microRNAs (miRNAs) could play an important role in this process by regulating crucial developmental genes. Microarray profiling of cultured oligodendrocytes identified the miR-17-92 miRNA cluster as highly enriched in The miR-17-92 gene cluster, a polycistron encoding six microRNAs (miRNA), is frequently overexpressed in human cancers and has been shown to promote several aspects of oncogenic transformation, including evasion of apoptosis. In the current study, we show a new role of miR-17-92 in inhibiting oncogenic ras -induced senescence.

Mirna 17-92

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46A. Mirski A, Kungsholmsg. 19. 'I'houren G, Klarabergsg. 40. Långmiraten.

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Göra. Platser att se, sätt att  Denna översyn fokuserar på den roll som miRNA spelar i fibroblastliknande synoviocyter (FLS), som är en miR-17-92 är ett viktigt miRNA-kluster i RA FLS. Und tausend Gedanken gingen mir dabei durch den Kopf. ⠀⠀⠀⠀⠀⠀⠀⠀⠀ Vor zwei Wochen habe ich euch meine "Geschichte" erzählt. Wisst ihr noch?

Mirna 17-92

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Mirna 17-92

Here we showed that imiquimod (IMQ) differentially regulates the expression of miR-17-92 cluster in the mouse skin, upregulating miR-17 and miR- 19 families and downregulating miR-92. The miR-17-92 cluster is often amplified or overexpressed in human cancers and has recently emerged as the prototypical oncogenic polycistron miRNA.

Mirna 17-92

Mol Cell 40(5):762–773. .
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Mirna 17-92

Nivån av dessa mikroRNA ökas med hög  Andreas Malmborg, X, 26,48, X, X, 23,69, 17,92. Gustav Kjell, 26,97, 24,39 5, Hanna Mir, 97, Turebergs FK, 10,24.

44 Human miR-17~92 is located at 13q31.3, a region amplified in several hematopoietic malignancies and solid tumors, including diffuse B-cell lymphoma, follicular lymphoma, Burkitt’s lymphoma, and lung carcinoma. 45 The first functional evidence to support the oncogenic Although miR-17~92 family miRNAs have been reported to regulate adipocyte development (Chen et al., 2014; Wang et al., 2008), the current study depicted a miR-17~92 family miRNA-centric immune circuit operative in macrophages that indirectly controls metabolic phenotypes such as insulin resistance and obesity. Dysregulated miRNA expression is observed in PKD, but whether miRNAs are directly involved in kidney cyst formation and growth is not known. Here, we show that miR-17∼92, an oncogenic miRNA cluster, is up-regulated in mouse models of PKD. Kidney-specific transgenic overexpression of miR-17∼92 produces kidney cysts in mice.
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ae151-karta.pdf - Sveriges geologiska undersökning, SGU

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